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Earlier this year, a research team at the Cleveland Clinic offered another explanation as to why meat intake may be related to mortality (see, for example, Harvards Meat and Mortality Studies )
This review summarizes glucose metabolism in the normal heart and highlights the features and regulatory mechanisms of glucose metabolic reprogramming in myocarditis, including the hypoxia-inducible factor-1/mammalian target of rapamycin axis, nuclear factor erythroid 2-related factor 2-mediated pentose phosphate pathway, immune-responsive gene 1/itaconate axis, and phosphoglycerate kinase 1
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