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Previous genetic studies have shown that conditional knockout of Ctnnb1 in the skin impedes the transition of hair follicles from the resting phase to the growth phase, leading to arrested hair growth ( Ctnnb1 is significantly upregulated in anwuligan-treated HFSCs, but this effect was not observed in skin tissues

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These results here agreed with the significant decrease in serum AST, urea, and creatinine in the SEC-treated group with bee venom compared to the positive control group, where these findings were in line with other previous studies [55, 61]

Inflammatory Response By invigorating the immune system, B12 might lead to a temporary spike in inflammation, which is essentially a marker that healing mechanisms are in play