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Moreover, in both groups, we previously described more numerous -SMA-positive myofibroblasts and blood vessels that were the typical symptoms of initial fibrosis, the long-term consequence of chronic inflammation in lung tissue caused by fibroblast proliferation and excessive collagen deposition (33, 42, 43)

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Afterward, this response continues to Th2 response in chronic HP and promotes inflammatory responses and fibrosis [185]