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Thus, chemotherapeutic approaches to blocking DNA replication through disruption of disulfide reductase pathways will need to be cognizant of the potentially complete functional redundancy of the GSH and Trx pathways in animal systems, and sensitive to the various other physiological roles these pathways play in normal homeostatic and stress-response pathways

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acknowledges support from the NIH (F31CA236372)

Protocol for a nested case-control study design for omics investigations in the Environmental Determinants of Islet Autoimmunity cohort