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- when there is risk of torsades de pointes, for example in patients with prolonged QT, concomitant administration of other medications that produce torsades de pointes (amiodarone, quinidine, bepridil, IV erythromycin, halofrantine, pentamidine, sparfloxacin, sultopride, vincamine, betrilio, disopyramide, sotalol, etc)

These include reduced neuroinflammation, decreased oxidative stress, enhanced neuronal survival, and improved synaptic plasticity

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153,154 Under conditions of insufficient glycolysis caused by glucose deprivation, 2-deoxyglucose treatment or Akt signaling inhibition, glutamine-addicted cells are more sensitive to GDH1 deficiency