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Abstract Fungal infections pose a significant threat to human health, particularly in immunocompromised individuals, driving a sustained increase in the demand for effective antifungal agents

It is structurally derived from p -aminophenol (PAP), a potent inducer of kidney toxicity

[23] described a few properties of Thermosynechococcus elongatus BP-1 (TeGST) and Synechococcus elongatus PCC 6301 GSTs (SeGST)

This amplification effect is why interactions involving enzyme inhibitors (such as certain antifungal drugs or even grapefruit juice) can lead to dramatic and dangerous increases in the plasma concentrations of co-administered high-extraction drugs

Some drugs are extensively metabolised during this process and this can decrease the bioavailability of the drug (which we will now discuss)