When we eat, glucose in our bloodstream triggers GLP-1 to release insulin
This is not theoretical speculation
GLP-1 reduces intracellular ROS, prevents oxidative stress injury, and increases cellular protection in arterial endothelial and smooth muscle cells [70, 71]
The most common adverse effects are gastrointestinal in nature, reflecting the medication's mechanism of action on GLP-1 and GIP receptors in the digestive system
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