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First, directionality diverges for METTL3 across cancer types: in CRC models it amplifies chemokines linked to myeloid influx, while in endometrial tumors it preserves NLRC5-driven MHC-I and correlates with higher CD8 + contentdifferences likely rooted in target selection and reader competition that are not yet mapped at base-resolution in primary human specimens
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10.1111/J.1582-4934.2010.01137.X 174
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