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On the one hand, mitochondria are the source of many danger signals released by dying cancer cells, and these signals are crucial for the DCs activation (Porporato et al., 2018) and tumor-associated macrophages (TAMs) (Icard et al., 2018), as well as the concentrations of succinate and citrate

The added GIP activity appears to synergistically amplify metabolic effects beyond GLP1 alone, contributing to superior weight reduction observed in clinical trials [3] [4]

Notably, the knockout of HDAC6 partially blocks the upregulation of TFEB downstream gene expression induced by TSA, indicating that the effect of TSA on TFEB activation depends on the inhibition of HDAC6 (Li X