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The expression and distribution of FFARs such as GPR40, GPR119, and GPR120 in L and cells and glucose, along with their dual effects on insulin and GLP-1 secretion, have made them attractive therapeutic targets, leading to the development of agonists for these receptors for the treatment of T2DM [150,151,152,153,154]

[PubMed ID: 11377818]

Individuals with metabolic syndrome features that span multiple domains (visceral obesity, insulin resistance, dyslipidemia) who want comprehensive metabolic intervention

In contrast, platelet-rich plasma (PRP) therapy has emerged as a regenerative treatment that enhances cartilage repair, reduces inflammation, and slows OA progression