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Patients with specific genetic predispositions in GLP-1R, GIPR, and FTO pathways may experience different efficacy trajectories, making genetic context essential for informed cost-benefit analysis and provider conversations about treatment selection

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Although NADPH has multifunctional roles in regulating inflammation, particularly in myeloid cells that have been clearly defined in the past, it remains unclear how NADPH-dependent inflammatory and anti-inflammatory pathways are co-regulated in order to fine-tune the magnitude of an inflammatory response