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The recruited macrophages are proinflammatory in character and conventionally classified as M1 type macrophages, whereas the M2 type macrophages resident in normal adipose tissue are anti-inflammatory and protective [46]

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Accordingly, the probable mechanisms could be discussed in four main approaches, namely, modulation of gut microbiota dysbiosis, production of SCFAs, reduction of gut permeability and metabolic endotoxemia, and regulation of bile acid metabolism (32, 35, 36)