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TNBC cells can express high levels of the xCT cystine/glutamate antiporter, which leads to a reduction in xCT-associated glutathione levels, thus reducing cell viability and sensitizing the cells to ferroptosis (13)

Evidence for oxidative/nitrosative stress in the pathogenesis of hepatic encephalopathy

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127 SFAs, such as palmitic acid, can increase the production of ceramide and NO, leading to impaired insulin synthesis, and the apoptosis of cells through inflammation, ERS, and oxidative stress, which can be blocked by unsaturated fatty acids

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